https://uoeldcu.org/site/uploads/docs/Dead%20Doctors%20Dont%20Lie%20by%20Joel%20Wallach%20(z-lib.org).pdf
Selenium Deficiency Diseases
Direct Results
Anemia
“Age spots” & “Liver spots” Fatigue
Muscular weakness
Myalgia (muscle pain and soreness)
Fibromyalgia
Scoliosis
Muscular dystrophy (MD, White Muscle Disease, Stiff Lamb Disease)
Cardiomyopathy (Keshan Disease, “Mulberry heart” Disease)
Heart palpitations
Atrial fibrilation
Liver cirrhosis
Pancreatitis
Pancreatic atrophy
Infertility
Low birth weight
High infant mortality
SIDS (Sudden Infant Death Syndrome)
Cystic Fibrosis (congenital)
Indirect Results
HIV (increased rate of conversion to AIDS and transmission to fetus) ALS
(Lou Gehrig’s Disease) MS (Multiple sclerosis) Alzheimer’s Disease
Cancer (increases cancer risk significantly)
Selenium deficiency can result in infertility in both men and women.
Congenital selenium deficiency during pregnancy can result in a wide
variety of problems ranging from miscarriage, low birth weight, high infant
mortality, cystic fibrosis, muscular dystrophy, cardiomyopathy, and liver
cirrhosis. Selenium deficiency in growing children can result in crib death
or SIDS (Sudden Infant Death Syndrome). Sixty-five percent of SIDS
deaths occur in children on canned infant formulas. Slow growth, small size
(failure to reach genetic potential for size and mass), muscular dystrophy,
scoliosis, cardiomyopathy (muscular dystrophy of the heart muscle or
Keshan Disease), anemia, liver cirrhosis, muscular weakness,
Appendix A-265
lowered immune capacity, and neuromuscular diseases are also linked to Se
deficiency.
In young adults, selenium deficiency appears as anemia, chronic fatigue,
muscular weakness, myalgia, fibromyalgia, muscular tenderness,
pancreatitis, infertility, muscular dystrophy, scoliosis, and cardiomyopathy.
Cardiomyopathy is quite common in young athletes such as basketball and
football players at the high school, college, university, and professional
levels because of Se deficiency, as are multiple sclerosis and liver cirrhosis.
Selenium deficiency in adults appears as reduced immune capacity, anemia,
infertility, “age spots” or “liver spots”, myalgia, fibromyalgia, muscle
weakness, MS, ALS, Parkinson’s Disease, Alzheimer’s Disease,
palpitations or irregular heart beat, cardiomyopathy, liver cirrhosis, and
cancer.
In a review of the anti-cancer effects of selenium, Dr. Gerhard N.
Schrauzer, head of the Department of Chemistry, UCSD, states:
Selenium is increasingly recognized as a versatile anticar-cinogenic agent.
Its protective functions cannot be solely attributed to the action of
glutathione peroxidase. Instead, selenium appears to operate by several
mechanisms, depending on dosage and chemical form of selenium and the
nature of the carcinogenic stress. In a major protective function, selenium is
proposed to prevent the malignant transformation of cells by acting as a
“redox switch” in the activation-inactivation of cellular growth factors and
other functional proteins through the catalysis of oxidation-reduction
reactions of critical -SH groups or -S-S- bonds.
The growth-modulatory effects of selenium are dependent on the levels of
intracellular glutathione peroxidase and the oxygen supply. In general,
growth inhibition is achieved by the Se-mediated stimulation of cellular
respiration (more oxygen, less cancer). Selenium appears to inhibit the
replication of tumor viruses in animals and the activation of oncogenes by
similar mechanisms. However, it may also alter carcinogen metabolism and
protect DNA against carcinogen-induced damage. In additional functions of
relevance to its anticar-cinogenic activity, selenium acts as an acceptor of
biogenic methyl groups, and is involved in detoxification of metals and
certain xenobiotics. Selenium also has immunopotentiating properties. It is
required for optimal macrophage and natural killer cell functions.
266 - DEAD DOCTORS DON’T DE
The school of pharmacy from the University of Georgia released a report in
August of 1994 that concludes that a human selenium deficiency is related
to the clinical onset of full blown AIDS in chronically infected HIV
patients. According to their report, HIV requires large amounts of selenium
for replication. In selenium deficient patients, the virus competes with the
patient for the limited amounts of available selenium. The HIV patient
actually dies of a chronic selenium deficiency encephalopathy, liver
cirrhosis, or cardiomyopathy. Long-term HIV patients (20 years or more)
that never developed full blown clinical AIDS had supplemented with large
amounts of selenium.
Clinical Diseases Associated with Vanadium Deficiency
Slow growth
Increased infant mortality Infertility
Elevated cholesterol
Elevated triglycerides
Hypoglycemia
Hyperinsulinemia
Narcolepsy
Prediabetes
Diabetes
ADD, ADHD
Depression
Manic depression, Bi-polar disease Tourette’s syndrome Cardiovascular
disease Obesity
Symptoms of Nickel Deficiency
( Poor growth
< Anemia
< Depressed oxidative ability of the liver
< Increased newborn mortality ( Rough/dry hair
( Dermatitis
( Delayed puberty
( Poor zinc absorption
Appendix A - 255
Less than 10 percent of ingested metallic nickel is absorbed. Nickel
deficiency was first reported in 1970. Nickel functions as a cofactor for
metalloenzymes and facilitates gastrointestinal absorption of iron and zinc.
Optimal tissue levels of vitamin B12 are necessary for the optimal
biological function of nickel. Vitamin B12 deficiency results in an increased
need for nickel by animals and man.
Deficiency Diseases of Manganese
Congenital ataxia
Deafness (malformation of otolithes) Asthma
Chondromalacia Chondrodystrophy “Slipped Tendon”
Defects of chondroitin sulfate metabolism (poor cartilage
formation) TMJ
Repetitive motion syndrome Carpal tunnel syndrome Convulsions
Infertility (failure to ovulate, testicular atrophy)
Still births/spontaneous abortions (miscarriages)
Loss of libido in males and females
Retarded growth rat