>>145288
>adhd but that's just a jewish myth
It's not a myth. It's sometimes a T3 thyroid hormone deficiency with adrenal compensation, or at least additional T3 + fuel can overcome the issue even if it's not the direct cause. This is exacerbated by a society which becomes authoritarian when they can't get a problem child to settle down, so they do things like sugar restriction, which makes the problem worse by increasing adrenal activity and lowering thyroid activity.
Weiss, R. E., M. A. Stein, and S. Refetoff. Behavioral effects of liothyronine (L-T3) in children with attention deficit hyperactivity disorder in the presence and absence of resistance to thyroid hormone. Thyroid 7.3 (1997): 389-393.
https://doi.org/10.1089/thy.1997.7.389
>Evidence that the thyroid may play a role in the pathogenesis of attention deficit hyperactivity disorder (ADHD) comes from observations that 48% to 73% of children with the syndrome of resistance to thyroid hormone (RTH) have ADHD.
RTH, as I have read, can be caused by excess polyunsaturated fat in the diet/bodily tissues, but it can also happen when there is merely a poor ability to utilize glucose which polyunsaturated fat contributes to, as glucose metabolism is one of the main activities regulated by thyroid hormones, so even if there was a confounding factor inhibiting the usage of glucose, it is possible that they had been previously labeled "resistant to thyroid hormone" superficially since they didn't quite understand why thyroid hormones didn't cause some action. RTH might just mean resistant to stimulants in general, but not that stimulants can't work if a high enough dose is given.
>[…]
>A prospective, randomized, double-blinded, placebo-controlled, cross-over study was conducted to evaluate the effect of the rapid acting thyroid hormone, liothyronine (L-T3), on the behavior of 8 children with ADHD + RTH, and 9 children with ADHD and normal thyroid function (ADHD Only).
The "normal" group might be thought of as not resistant to stimulants.
>L-T3 had no effect on Conners Hyperactivity Index in 7 of 9 children with ADHD Only; it caused improvement and deterioration in 1 subject each. In contrast, the rating in 5 of 8 subjects with ADHD + RTH showed improvement, whereas 3 of 8 subjects remained unchanged.
>L-T3 was associated with increased commission errors in 5 of 8 children with ADHD Only
Since they were probably already good at using glucose (due to being responsive to stimulating hormones) they probably didn't have much fuel laying around to burn when stimulated so their brains had less energy due to the subsequent drop in blood sugar.
>and decreased commission errors in 4 of 7 with ADHD + RTH.
I think the better success rate in this group can be attributed to them being more likely to have fuel laying around to burn from the history of being resistant to thyroid hormones (or possibly resistant to stimulation in general) creating a backlog.
The study doesn't mention if the children were fasted or if they had eaten. The children who had positive results were more likely to have eaten more recently. Imagine giving strong coffee to someone on an empty stomach. T3 is a stimulant very similar to caffeine. Of course some of them got into adrenaline mode to compensate for the drop in blood sugar. Yes, it's an astounding result. ADHD can be treated with the use of stimulants and of course simultaneous administration of food providing carbohydrates in order to sustain blood glucose. The ADHD child is wild because he lacks energy (absence of glucose which raises adrenaline to provide animal energy but not human energy) or is distracted because he can't put it to good use (by use of thyroid hormones).
Boelen, Anita. Thyroid hormones and glucose metabolism: the story begins before birth. Experimental physiology 94.10 (2009): 1050-1051.
https://doi.org/10.1113/expphysiol.2009.049361
Arnold, L. Eugene. Alternative treatments for adults with attention‐deficit hyperactivity disorder (ADHD). Annals of the New York Academy of Sciences 931.1 (2001): 310-341.
https://doi.org/10.1111/j.1749-6632.2001.tb05788.x
>Zinc supplementation is hypothetically supported by systematic case‐control data, but no systematic clinical trial.
…
>Simple sugar restriction seems ineffective.
…
>Thyroid treatment is effective in the presence of documented thyroid abnormality.
Wong, Rosemary, et al. Transgenic mice bearing a human mutant thyroid hormone beta 1 receptor manifest thyroid function anomalies, weight reduction, and hyperactivity. Molecular Medicine 3.5 (1997): 303.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2230075/
Herein transgenic mice lose T3-binding and transcriptional activation and become hyperactive. Some human studies say hyperthyroidism is ADHD, but what I have seen when I examine these studies in detail is a high level of T4 but a failure to transform it to T3 which is effectively still hypothyroidism.
Alternatively, hyperactivity and not paying attention to nonsense in a certain context could merely be health and intelligence, and slow people are easily annoyed pricks and fatasses.