Glutamate impairs the NMDA receptors ability to function https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5098640/
What DXM does is allow the NMDA receptors to rewire in a healthy fashion and replenish some shit that schizo brains don't have.
We don't know what causes schizophrenia so we don't know what cures it but
>CYP2D6 ultrarapid metabolization
which can be caused by DXM, is a possiblity as well.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3298719/
>Overall, there is accumulating evidence, albeit mostly adjunctive treatments, that agents working on inflammatory pathways have some benefits in people with schizophrenia.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3641824/
>plasma levels of IL-1β and TNF-α were significantly higher in patients with schizophrenia than in controls, but plasma BDNF levels were significantly lower. Patients were treated with the atypical antipsychotic risperidone (Risp) only or with Risp+add-on dextromethorphan (DM). PANSS scores and plasma IL-1β levels significantly decreased, but plasma TNF-α and BDNF levels significantly increased after 11 weeks of Risp treatment. Patients in the Risp+DM group showed a greater and earlier reduction of symptoms than did those in the Risp-only group. Moreover, Risp+DM treatment attenuated Risp-induced plasma increases in TNF-α.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3611097/
> it is likely that donepezil would (an NMDA antagonist like DXM) be potential therapeutic drugs for the treatment of the cognitive deficits in schizophrenia.
https://www.ncbi.nlm.nih.gov/pubmed/19433073
But neither of you bothered to Google its effects did you?
XXDDD CIAnigger get out lmao.
Get real, you're both here to laugh at Terry.