>>128897
It is possible that boosting the oxidative metabolism may help. Let's consider how some researchers treat ADHD. You didn't say you had ADHD, but whether or not you have a clinical diagnosis may or may not be relevant. Few ailments are black and white, on/off switches. There is usually a continuous spectrum of conditions, such as obesity before diabetes, eccentricity before insanity, etc. What makes a diabetic man merely obese, may make the merely obese man a bodybuilder if they happen to suffer from the same problems, only in differing severity.
Behavioral effects of liothyronine (L-T3) in children with attention deficit hyperactivity disorder in the presence and absence of resistance to thyroid hormone.
>Evidence that the thyroid may play a role in the pathogenesis of attention deficit hyperactivity disorder (ADHD) comes from observations that 48% to 73% of children with the syndrome of resistance to thyroid hormone (RTH) have ADHD.
[…]
>In children with RTH and ADHD, particularly those that exhibit hyperactivity, L-T3 in supraphysiological doses may be beneficial in reducing hyperactivity and impulsivity. In the majority of children with ADHD who do not have RTH, L-T3 treatment has no effect or may be detrimental.
https://www.ncbi.nlm.nih.gov/pubmed/9226208
Chronic caffeine treatment during prepubertal period confers long-term cognitive benefits in adult spontaneously hypertensive rats (SHR), an animal model of attention deficit hyperactivity disorder (ADHD).
>The spontaneously hypertensive rat (SHR) is frequently used as an experimental model for the study of attention deficit hyperactivity disorder (ADHD) since it displays behavioural and neurochemical features of ADHD.
[…]
>These findings suggest that chronic caffeine treatment during prepubertal period confers long-term cognitive benefits in discriminative learning impairments of SHR, suggesting caffeine as an alternative therapeutic strategy for the early management of ADHD symptoms.
https://www.ncbi.nlm.nih.gov/pubmed/20600342
So it seems both caffeine and T3 have the potential to treat ADHD in at least some contexts, but why and what could these substances even be doing? It turns out that caffeine is thyroid-mimetic, and T3 is the active thyroid hormone. Both of these substances boost the oxidative metabolism that makes proper and efficient use of glucose, and the brain is a glucose hungry organ. This hints at the idea that you might be able to increase your attention span by administering any substance that boosts the oxidative metabolism. Thiamine (vitamin B1), niacinamide, aspirin, and methylene blue( are all likely to be helpful. In fact, caffeine and aspirin are more effective together than either alone based on the research I have read and my own experiences.
Since I am only taking a cursory glance at the literature on ADHD treatments, I can only offer conjecture, but I suspect that the T3 treatment on ADHD children wasn't as effective as it should have been had the researchers known the most beneficial context in which to administer T3. You have to eat adequate carbs, preferably sugar with many secondary nutrients to get the right results from T3 or caffeine. If you don't supply a nutritious meal or carbs with T3, what is likely to happen is a surge of cortisol and adrenaline, which would easily explain detrimental effects in some subjects, but it is "common sense" to restrict sugar intake in ADHD children. Actually, it seems to be the standard practice to constantly micro manage such children and exert tyrannical authority and overbearing demands on them as they are often seen as wild and unruly. Such children may in fact be trying to treat their condition by gulping down sweets whenever they find them as in order to burn glucose properly you have to, well, have glucose for starts. I would suggest that sugar is in fact an essential part of treating conditions such as ADHD, but few will interpret this bare statement the right way as I don't mean pure nutritionless sugar is a cure. Frequent orange juice is a good idea as it supplies both sugar and thiamine which is essential to the oxidative metabolism, but any diet that supplies a wide range of vitamins and minerals such as one that includes liver, egg yolks, and low fat seafood can get away with a hefty amount of supplemental table sugar.
Polyunsaturated fats work against the actions of thyroid hormone both in its creation, for example by creating fatty liver disease (Most of T3 is made in the liver.), and its uptake by cells, and they accumulate over time in tissues throughout the body causing progressive aging and increasing hypothyroidism through continued consumption. So if T3 treats ADHD, then a diet with less polyunsaturated fats also treats it, but so would lowering intake of all fats as they all have the potential to steer metabolism away from the efficient and proper oxidative metabolism of glucose.